Canonical Oncology Mechanisms

What if the therapeutic mechanism of Nanomedicine ZPH belonged to none of these — but to an entirely different system?

1- Blocks the PD-1 receptor on T cells, enhancing the immune system's ability to attack cancer cells.
2- Inhibits cyclin-dependent kinases 4 and 6, preventing cell cycle progression and tumor growth.
3- Blocks tyrosine kinase enzymes, interrupting signaling pathways that promote cancer cell growth.
4- Binds to specific antigens on cancer cells, marking them for destruction by the immune system.
5- Blocks the proteasome, leading to the accumulation of proteins that induce cancer cell death.
6- Inhibits poly (ADP-ribose) polymerase, preventing DNA repair in cancer cells.
7- Inhibits the formation of new blood vessels that supply the tumor with nutrients.
8- Inhibits topoisomerase enzymes, preventing DNA replication and transcription.
9- Disrupts microtubule function, inhibiting cell division.
10- Blocks or modulates hormone receptors, slowing the growth of hormone-sensitive tumors.
11- Blocks the BCL-2 protein, promoting apoptosis in cancer cells.
12- Inhibits histone deacetylases, altering gene expression to inhibit cancer cell growth.
13- Uses genetically modified viruses to selectively infect and kill cancer cells.
14- Enhances the patient's own immune cells to better attack cancer cells.
15- Alters epigenetic marks to change the expression of genes involved in cancer.
16- Blocks vascular endothelial growth factor, reducing blood supply to the tumor.
17- Inhibits DNA methyltransferases, leading to reactivation of tumor suppressor genes.
18- Modulates the immune system to enhance its anti-cancer activity.
19- Genetically engineers T cells to express receptors specific to cancer cell antigens.
20- Inhibits checkpoint kinases, disrupting the cell cycle in cancer cells.